How to treat heart disease without a transplant

A group of doctors have developed a treatment that could help treat heart failure, a rare but deadly condition.

Researchers from the University of Leeds in the United Kingdom have discovered a new type of protein that may help control the progression of heart failure.

Heart failure can occur when the body does not receive enough blood to supply the heart with oxygen.

It is the leading cause of death in the UK and is the most common type of heart attack.

People with heart failure often suffer for years from fatigue, poor blood circulation, heart attacks and other problems.

A new study published in the Journal of Clinical Endocrinology & Metabolism showed that the protein called apoe3 was able to stop the progression to heart failure by blocking the breakdown of proteins that normally help regulate the body’s heart.

This protein is also found in other body parts, like the lungs and skin.

The researchers have found that apoe2, a protein in the skin, can be used to help the body break down protein breakdown.

This is the first time that a new protein has been found to stop heart failure in people with heart disease.

The discovery could lead to new drugs that could be used as treatments for the condition.

The new protein was also able to reverse the damage caused by the heart failure-related disease of apoea4.

It was found that the new protein could be produced by a patient’s own body or by a donor.

It’s unclear whether the new treatment would work in people who have heart failure and do not have the disease, or whether it could be adapted to other types of heart disease and be used in people without heart failure as a way to control the disease.

Dr Richard Tait, of the University’s Department of Medicine, said: ‘This study shows how a new way of producing proteins that control the body and help it break down proteins that are normally responsible for controlling blood supply to the heart can also help with the progression from heart failure to heart disease.’